Jump to content

Talk:Virus

Page contents not supported in other languages.
From Wikipedia, the free encyclopedia
Featured articleVirus is a featured article; it (or a previous version of it) has been identified as one of the best articles produced by the Wikipedia community. Even so, if you can update or improve it, please do so.
Main Page trophyThis article appeared on Wikipedia's Main Page as Today's featured article on October 5, 2009.
Article milestones
DateProcessResult
June 20, 2006Peer reviewReviewed
July 14, 2006Featured article candidateNot promoted
October 19, 2006Good article nomineeListed
November 4, 2007Featured article candidateNot promoted
February 6, 2008Good article reassessmentKept
September 23, 2008Featured article candidatePromoted
Current status: Featured article


Mention of viruses as obligate parasites?

[edit]

Just read a paper on the topic, and wondered if it would fit here. I try not to make content changes on larger pages without consulting the talk page first. FullyNatural (talk) 20:07, 9 April 2024 (UTC)[reply]

This is pretty much covered in the first sentence. Viruses aren't traditionally thought of as parasites as they are dependent on the host (cell) for much more than just nutrients. What was the paper? Graham Beards (talk) 10:14, 10 April 2024 (UTC)[reply]
Here's the DOI link: https://doi.org/10.1016%2FB978-012373944-5.00323-0
You're right in saying viruses aren't typically thought of as viruses. FullyNatural (talk) 13:35, 12 April 2024 (UTC)[reply]
That might be the stupidest thing I've ever written. Of course I mean "parasites" not "viruses"... FullyNatural (talk) 13:41, 12 April 2024 (UTC)[reply]
That 2009 article looks like it was based on an earlier version of our article [1] and is not a good source. I don't think it would help our readers to add "obligate intracellular parasites" - it confuses more than it informs. Graham Beards (talk) 13:48, 12 April 2024 (UTC)[reply]
Fair point, thanks for the insight! FullyNatural (talk) 19:09, 12 April 2024 (UTC)[reply]

Suggestion: add history of virology to the article

[edit]

Is there a good reason why History of virology doesn't appear here? Vonfraginoff (talk) 15:07, 7 July 2024 (UTC)[reply]

Good point. It used to be part of this article but it was too long and given it's own article. I'll write a summary and leave a {{main|History of virology}}. Graham Beards (talk) 06:47, 8 July 2024 (UTC)[reply]
Looks great, many thanks! Vonfraginoff (talk) 03:49, 1 August 2024 (UTC)[reply]

Recent edits

[edit]

I have removed two recent edits. The first one, on the bubble theory, was cited to a single source that presents a hypothesis that has not yet been accepted by the scientific community and as such is a primary source. The second on life properties of viruses, added a little to what has already been said, and what on earth is a "mare spore"? The added text also suffered from padding, editorializing and below par prose. Graham Beards (talk) 11:00, 8 July 2024 (UTC)[reply]

Replace illustration

[edit]

I have replaced this illustration File:Influenza geneticshift.svg Reason:

  • It is an inaccurate reproduction of the source [1]. The two "parents" are correctly labeled, but in the original the progeny is not given a description whereas the Wikimedia reproduction labels the progeny as "highly pathogenic human strain". Accompanying text in the original explains that a human strain may acquire characteristics from a highly pathogenic avian strain, but makes no assumption about the characteristics of the progeny.

Bob (talk) 17:07, 17 July 2024 (UTC)[reply]

The original diagram dates from 2007 and it was not taken from the 2014 source! The cartoon was just a schematic used to illustrate reassortment. I think you should retract your comments here and at the Commons, but the new image you have added can stay IMHO. Graham Beards (talk) 17:58, 18 July 2024 (UTC)[reply]

References

  1. ^ Yoo E (February 2014). "Conformation and Linkage Studies of Specific Oligosaccharides Related to H1N1, H5N1, and Human Flu for Developing the Second Tamiflu". Biomolecules & Therapeutics. 22 (2): 93–99. doi:10.4062/biomolther.2014.005. PMC 3975476. PMID 24753813.

Semi-protected edit request on 4 October 2024

[edit]

Recent work identified metabolic interference in the innate defense against viruses. Human hepatocytes were found to activate both PPARA and FXR nuclear receptors, hindering HCV replication and assembly, as a metabolic defense mechanism to limit viral growth. [1] Yaakov Nahmias (talk) 20:53, 4 October 2024 (UTC)[reply]

 Not done: it's not clear what changes you want to be made. Please mention the specific changes in a "change X to Y" format and provide a reliable source if appropriate. Also seems way too focused for this article Cannolis (talk) 00:51, 5 October 2024 (UTC)[reply]

Virus

[edit]

Show the diagrams of poliomyelitis virus, tobacco mosaic virus, bacteriophage 102.91.4.29 (talk) 18:10, 9 October 2024 (UTC)[reply]

Commons: poliomyelitis virus, tobacco mosaic virus, bacteriophage T4. --Petr Karel (talk) 07:25, 10 October 2024 (UTC)[reply]

The redirect Virose has been listed at redirects for discussion to determine whether its use and function meets the redirect guidelines. Readers of this page are welcome to comment on this redirect at Wikipedia:Redirects for discussion/Log/2024 December 1 § Virose until a consensus is reached. Plantdrew (talk) 01:52, 1 December 2024 (UTC)[reply]

The redirect Virus' has been listed at redirects for discussion to determine whether its use and function meets the redirect guidelines. Readers of this page are welcome to comment on this redirect at Wikipedia:Redirects for discussion/Log/2024 December 1 § Virus' until a consensus is reached. Plantdrew (talk) 01:53, 1 December 2024 (UTC)[reply]

  1. ^ Levy, Gahl; Habib, Naomi; Guzzardi, Maria Angela; Kitsberg, Daniel; Bomze, David; Ezra, Elishai; Uygun, Basak E.; Uygun, Korkut; Trippler, Martin; Schlaak, Joerg F.; Shibolet, Oren; Sklan, Ella H.; Cohen, Merav; Timm, Joerg; Friedman, Nir; Nahmias, Yaakov (2016). "Nuclear receptors control pro-viral and antiviral metabolic responses to hepatitis C virus infection". Nature Chemical Biology. Nature Publishing Group. doi:10.1038/NCHEMBIO.2193.